Recent diabetic retinopathy

I am in the case that you have diagnosed a very mild diagnostic retinopathy for a few months.I'm baking some sport and eating healthy and I'm very controlled.

Hello.I also have non -proliferative retinopathy.I am diabetics 25 years and I have 33 I diagnosed a very mild retinopathy principle at 27 but it would make me more and a little inflamed the macula of the right eye.And basically it has been my fault when I was younger, for not taking diabetes seriously, and always having the glycosylated above 8.o minimum 7.8.It scares when they diagnose it, I'm still scared waiting if it can go more or not, I'm going every 6 months.And working to lower my glycosilada.It helps to read that you are not the only one, because if diabetes is already hard (at least in my case) go adding ... in short, thank you for compatting your stories, they help to raise awareness and see what is possible to stop it.A hug.

Thank you Javier Arriaga Sanz, paying attention to him, it was my ophthalmologist for many years and now he is a partner of his.Obviously the ophthalmologist who sees you has to be a specialist in retina and diabetes, it is so.Normally in all hospitals there are and the endocrine derive us, at least in my case it was so.I am type 1 diabetic since the age of 10 and I have almost 49, so about 40 years of diabetes.As you have said many, my control was not very good for several years in adolescence and besides, when I started there was not even because of today, today's control techniques, the glucometers were bumps and very inaccurate, they told you that you would measure youOnce a day, insulins were pork (q is the most similar to the human) and with roads and syringes.Today with continuous measurement sensors and insulins that are, nothing to see.
I think that I have told my story once but to abbreviate I will say that I am a family doctor and the 18 or 19 to approximately, I was diagnosed with microaneurisms and hemorrhages in both eyes with perfect vision.Although it did not become a retinop.Proliferative, (if we wait for that, it can be late), the ophthalmologist decided that it had to be aggressive and panfotocoagular.I am Panfotocoagulated for almost 20 years I still maintain good vision and I am fine, except for a macular cyst that goes and goes when you want and that in the last two reviews it has not come down and it will be seen if I need treatment.By this I mean, first, that with the media today, retinopathy when it appears, it will be milder if there is general good control, and q, like everything in medicine, early detection would see the prognosis much, so there is if there areAlterations even if they are small, we will have to review them every less time than before and treat them if necessary.

Hello!I have been with diabetes for 33 years ... I have always gone annually or every 6 months to the ophthalmologist (which I assure you are specialized).He will do 6 years, my ophthalmologist told me that I had retinopathy and that same day gave me laser in both eyes.Panfotocoagular.I crumbled 100%.

Since then I go to review every 6 months and everything is going "well."It watches me some neovants that I have not to give it a laser ... Whenever I am going to review and return from it, I get rid of me ... I think what will be of me ...

As many, I spent a time when there was not so much control and that has taken me an invoice ... I was just detected the retinopathy and I was already buying the sensors on my own, which I suppose I will have helped me infinity ...Because the glyd I reduced her a lot (although I would like it even more).

My ophthalmologist tells me that "everything is going well."It is an issue that sometimes takes my dream and gives me a lot of anxiety.She knows ... and I don't know if that's why she doesn't tell me anymore.I don't think he omits me information.
He tells me that at the moment we are watching him because he doesn't want to give me more laser.

She knows that I am trying to lower the glyd to give me authorization for a pregnancy.He has never told me no ... and the endocrine either.Am I wasting time ...?

Apart from the laser there is an injections truth ...?Then, does it only have to operate ...?And if it doesn't work ... Is there no more ... right?😔
Is it true that you do what you do with the course of the disease always appears and develops ...?The only thing that can be stopped and postponed the progress?@meginer @"Javier Arriaga Sanz"

silviagrz said:
Hello!I have been with diabetes for 33 years ... I have always gone annually or every 6 months to the ophthalmologist (which I assure you are specialized).He will do 6 years, my ophthalmologist told me that I had retinopathy and that same day gave me laser in both eyes.Panfotocoagular.I crumbled 100%.

Since then I go to review every 6 months and everything is going "well."It watches me some neovants that I have not to give it a laser ... Whenever I am going to review and return from it, I get rid of me ... I think what will be of me ...

As many, I spent a time when there was not so much control and that has taken me an invoice ... I was just detected the retinopathy and I was already buying the sensors on my own, which I suppose I will have helped me infinity ...Because the glyd I reduced her a lot (although I would like it even more).

My ophthalmologist tells me that "everything is going well."It is an issue that sometimes takes my dream and gives me a lot of anxiety.She knows ... and I don't know if that's why she doesn't tell me anymore.I don't think he omits me information.
He tells me that at the moment we are watching him because he doesn't want to give me more laser.

She knows that I am trying to lower the glyd to give me authorization for a pregnancy.He has never told me no ... and the endocrine either.Am I wasting time ...?

Apart from the laser there is an injections truth ...?Then, does it only have to operate ...?And if it doesn't work ... Is there no more ... right?😔
Is it true that you do what you do with the course of the disease always appears and develops ...?The only thing that can be stopped and postponed the progress?@meginer @"Javier Arriaga Sanz"

For macular edema, there are intravitreal injections if this edema increases and can be affecting the vision, if it does not improve (after several sessions, it is not immediate), there is an intraocular device that releases corticosteroids in a delayed way for six months and I believethat the last thing is vitrectomy.
If you are Panfotocoagulated, you have burned the entire peripheral retina, you cannot have more laser sessions there, you have the macula for the central vision that when it is altered with an edema, it is when those injections are placed.
As for your pregnancy, better first comment with your endocrine and if you give you the approval of you, you reach the gyz.According to the status of that retinopathy, I imagine that they will advise you or not pregnancy.If your ophthalmologist knows that you are planning it and has not told you that no, I imagine that you can a priori.
And regarding whether you do what you do, we will have problems ... Well, daughter, in medicine nothing is accurate, many things influence and talk about risk factors.The first risk factor is the evolution time of the DB, obviously the more TPO, the more risk of problems, the second is the degree of control, the lower the gyted and lower glucemic variability the better.The first, it would not be so important today with the means of control that we have, you can have a very good control since debuts, when you and I started, that was unthinkable and I spent more than 10 years badly controlled, so I centerMore in the second factor because I can't change it, I will try to improve what is in my hand, I can't do something else ... and trust professionals and a little also in luck, there is no other.
I can not do more, I have a gyze of 5.5-5.6 with a very important effort and dedication so we trust it.

@meginer thousand thousand really 😘

silviagrz said:
@meginer thousand thousand really 😘

Then I write you a private

Hi Silvia.Your ophthalmologist has done what I owed, photocoagular, although I cannot agree with that "does not want to give me more laser."Actually, no one "wants" to give laser;or it is necessary, or it is not.And in your case, I think it is.If they remain neovosum (which are not directly photocoagulated), it is because there are still areas of anoxic retinal (not oxygenated), by photocoagular, and as guidance, the neovasos (except the papillary), are usually located at the edges of the areas of noPerfusion, and those will be easily detected with angiography, which I suppose that they haven't made you a long time.These anxic or non -perfusion zones are the stimulus of the appearance of neovants and their persistence, so while these areas are not eliminated by photocoagulation, neovas will persist with the consequent risk of hemorrhage and glial proliferation.
These are basic concepts in diabetic retinopathy.I retired 9 years ago and therefore I am not very aware of the novelties, but as far as I know, intravitee injections constitute a purely symptomatic treatment.Given the presence of the VEGF, an anti -GFF product is injected, and of course, by the action of the medicine disappears, but the causative factor of the vegf is not eliminated that is none other than the ischemia of the areas that have produced the neovosses, and therefore, injections are always necessary.
I am sorry to give you this news, but this is a very important struggle, and the more information you have more possibilities you will have to win.Greetings

@"Javieroftalmologist" in 6 months I have an appointment again.I will tell you all this.
My ophthalmologist is "far on me" and I don't "leave it" either, I prefer to know and if you can act.It's something that worries me a lot ... you don't know how much level.
I imagine that I will not have left me free will ... and that there is no imminent risk ...

javieroftalmologist said:
Hi Silvia.Your ophthalmologist has done what I owed, photocoagular, although I cannot agree with that "does not want to give me more laser."Actually, no one "wants" to give laser;or it is necessary, or it is not.And in your case, I think it is.If they remain neovosum (which are not directly photocoagulated), it is because there are still areas of anoxic retinal (not oxygenated), by photocoagular, and as guidance, the neovasos (except the papillary), are usually located at the edges of the areas of noPerfusion, and those will be easily detected with angiography, which I suppose that they haven't made you a long time.These anxic or non -perfusion zones are the stimulus of the appearance of neovants and their persistence, so while these areas are not eliminated by photocoagulation, neovas will persist with the consequent risk of hemorrhage and glial proliferation.
These are basic concepts in diabetic retinopathy.I retired 9 years ago and therefore I am not very aware of the novelties, but as far as I know, intravitee injections constitute a purely symptomatic treatment.Given the presence of the VEGF, an anti -GFF product is injected, and of course, by the action of the medicine disappears, but the causative factor of the vegf is not eliminated that is none other than the ischemia of the areas that have produced the neovosses, and therefore, injections are always necessary.
I am sorry to give you this news, but this is a very important struggle, and the more information you have more possibilities you will have to win.Greetings

Hi Javier, we know each other and I guess you will know who I am.
Then the intravitreas do not solve edema?, I thought that.In fact they went very well two years ago, but it is true that it has increased again and they have programmed me again.
Is there a limit or when the vision is lowered by the edema has increased and is there?
Can they be put continuously without problem?Thanks and a hug.

@"Javier Arriaga Sanz" @"javieroftalmologist" My case is different, but it also affects my eyes, for the moment in the eye funds that make me have not yet detected problems in the retina, but for about two years my dm2 me me meIt has affected the pairs of cranial nerves and originated strabismus in my left eye.Strabismus wants me to operate but I have many objections because I think that although they correct this strabismus, my DM2 will continue to affect the cranial pairs and that will have no end.Although my problem is serious and I would like to solve it is afraid to get into a spiral of operations without the end of solving it.Can you guide me a little, which I am very lost?
Thank you so much

The most frequent affection in diabetics, in terms of cranial pairs, is that of the VI torque, or external ocular motor, and is usually transitory, although it is very annoying because it usually causes diplopia (double vision).If, in your case, it has stabilized and has become permanent, surgery could be considered.However, I advise you to consult with a strabismus specialist, which is the one that can value it better

,

meginer said:

javieroftalmologist said:
hello silvia.Your ophthalmologist has done what I owed, photocoagular, although I cannot agree with that "does not want to give me more laser."Actually, no one "wants" to give laser;or it is necessary, or it is not.And in your case, I think it is.If they remain neovosum (which are not directly photocoagulated), it is because there are still areas of anoxic retinal (not oxygenated), by photocoagular, and as guidance, the neovasos (except the papillary), are usually located at the edges of the areas of noPerfusion, and those will be easily detected with angiography, which I suppose that they haven't made you a long time.These anxic or non -perfusion zones are the stimulus of the appearance of neovants and their persistence, so while these areas are not eliminated by photocoagulation, neovas will persist with the consequent risk of hemorrhage and glial proliferation.
These are basic concepts in diabetic retinopathy.I retired 9 years ago and therefore I am not very aware of the novelties, but as far as I know, intravitee injections constitute a purely symptomatic treatment.Given the presence of the VEGF, an anti -GFF product is injected, and of course, by the action of the medicine disappears, but the causative factor of the vegf is not eliminated that is none other than the ischemia of the areas that have produced the neovosses, and therefore, injections are always necessary.
I am sorry to give you this news, but this is a very important struggle, and the more information you have more possibilities you will have to win.Greetings

Hi Javier, we know each other and I guess you will know who I am.
Then the intravitreas do not solve edema?, I thought that.In fact they went very well two years ago, but it is true that it has increased again and they have programmed me again.
Is there a limit or when the vision is lowered by the edema has increased and is there?
Can they be put continuously without problem?Thanks and a hug.

Of course, who are you.Intravitreas if edema resolve, but temporarily.That is, the presence of VEGF (endothelial vascular growth factor) is being fought with Antivegf, but cannot be prevented, at least for now, to reappear the vegf again.Over time the good medication tolerance has been proven, and complications are reduced to intraocular infections linked to injection mechanics.(You already know that risk 0 does not exist)
In your case, in addition, it constitutes the only option as it is a residual edema in a photocoagulated macula.
As you know, I am at your disposal, although a very competent ophthalmologist is treating you that can solve any questions much better than me.Kisses

javieroftalmologist said:
,

meginer said:

javierofthalmologist said:
javierofthalmologist said:
hello silvia.Your ophthalmologist has done what I owed, photocoagular, although I cannot agree with that "does not want to give me more laser."Actually, no one "wants" to give laser;or it is necessary, or it is not.And in your case, I think it is.If they remain neovosum (which are not directly photocoagulated), it is because there are still areas of anoxic retinal (not oxygenated), by photocoagular, and as guidance, the neovasos (except the papillary), are usually located at the edges of the areas of noPerfusion, and those will be easily detected with angiography, which I suppose that they haven't made you a long time.These anxic or non -perfusion zones are the stimulus of the appearance of neovants and their persistence, so while these areas are not eliminated by photocoagulation, neovas will persist with the consequent risk of hemorrhage and glial proliferation.
These are basic concepts in diabetic retinopathy.I retired 9 years ago and therefore I am not very aware of the novelties, but as far as I know, intravitee injections constitute a purely symptomatic treatment.Given the presence of the VEGF, an anti -GFF product is injected, and of course, by the action of the medicine disappears, but the causative factor of the vegf is not eliminated that is none other than the ischemia of the areas that have produced the neovosses, and therefore, injections are always necessary.
I am sorry to give you this news, but this is a very important struggle, and the more information you have more possibilities you will have to win.Greetings

Hi Javier, we know each other and I guess you will know who I am.
Then the intravitreas do not solve edema?, I thought that.In fact they went very well two years ago, but it is true that it has increased again and they have programmed me again.
Is there a limit or when the vision is lowered by the edema has increased and is there?
Can they be put continuously without problem?Thanks and a hug.

Of course, who are you.Intravitreas if edema resolve, but temporarily.That is, the presence of VEGF (endothelial vascular growth factor) is being fought with Antivegf, but cannot be prevented, at least for now, to reappear the vegf again.Over time the good medication tolerance has been proven, and complications are reduced to intraocular infections linked to injection mechanics.(You already know that risk 0 does not exist)
In your case, in addition, it constitutes the only option as it is a residual edema in a photocoagulated macula.
As you know, I am at your disposal, although a very competent ophthalmologist is treating you that can solve any questions much better than me.Kisses

Thank you very much, I am aware of it and happy with the follow -up, I have been lucky with both of them.
A strong hug